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Markus Grompe, MD Oregon Health Sciences University, Portland, Oregon In 1995 a group of researchers in Portland, Oregon mapped the Fanconi anemia group D gene to human chromosome 3p. Very recently, the region containing the gene was narrowed down to a very small area, which contained only 3 genes. All 3 genes were analyzed in detail and mutations were found in 2 patients with group D Fanconi anemia. The mutations proved that the FANCD gene had indeed been cloned. Interestingly, however, not all complementation group D patients had mutations in this gene. For example, the HSC62 cell line, used to define FA group D, does not have mutations in the new gene. It is therefore likely that FA complementation group D is heterogeneous and represents at least 2 distinct genes. Because the HSC62 cell is the reference cell line for group D, we termed the new gene FANCD2. Interestingly, many other multicellular organisms including plants, fruit flies, and worms have a FANCD2 homologue. This is in contrast to other FA genes isolated to date, which seem to exist only in vertebrates. The finding of a FANCD2 gene in these other organisms is potentially very important for the future study of the FA pathway, since these other organisms are comparatively easy to study genetically. The FANCD2 gene is very large and has 44 exons. The protein is also large and has 1, 451 amino acids. Dr. D'Andrea's lab in Boston has produced an antibody to FANCD2. The FANCD2 protein resides in the nucleus of cells and exists in 2 forms, termed FANCD2-S short ; and FANCD2-L long ; . Interestingly, cells from most 95% ; FA patients contain only the FANCD2-S form of the protein. Therefore, FA can be readily diagnosed by a rapid antibody test which determines whether both forms of the protein are present or not. Chromosome breakage analysis is not required for this test.
Citalopram Average Side Effect N 1063 Placebob Autonomic Dry mouth + + + Sweating + + + Central peripheral nervous system Headache NAc NA d + Sedatione + + + Aggression e 0 + Dizziness NAc NA d + Tremor -- + + + Gastrointestinal Nausea + + + Diarrhea + NA d Constipation NAc NAc + + + Dyspepsia + + + Vomiting + NA d NAc + + Abdominal pain + NAc NAc NAc NA + Musculoskeletal c c Myalgia + NA NA Psychiatric Somnolence + + + Insomnia + + + Asthenia NA + + Anorexia + + + Anxiety + + + Agitation + NA + NAc + + Manic switche 0 + + Extrapyramidal symptomse 0 + + Nervousness NAc + + + Decreased libido + + + Upper respiratory Upper respiratory infection + NA + NAc NA + Rhinitis + NAc NAc NA NA + Sinusitis + NAc NA NA NA Urogenital Ejaculation difficulty + 0 + Micturition problems NAc NA + + Impotence + + + General Drug interactionse + + + Dermatologic reactionse + + + Weight gaine + 0 + Weight loss e 0 + Hyponatremia e 0 + Discontinuation syndromee 0 + 0 Fatigue + NA NA Fever + NA NA NAc NA + a Scores are based on the frequency of effects that occurred in treatment and placebo patient populations cited in Physicians' Desk Reference, 54th ed., 2000.8 Percentages 1.0% were scored as 0 + ; from 1.0% to 5.0% as " + "; from 5.1% to 10.0% as " + "; from 10.1% to 15.0% as " + "; and 15.1% as " + "; -- data not avaliable. b Scores in this column represent treatment-emergent side effect frequencies in the placebo group, averaged across 5 SSRIs. Placebo incidences of side effects based on frequencies reported in the PDR 8 ; were totaled, and the mean is expressed as a percentage of a total N value, and then scored as described. Note: In the data composite, placebo rates differ, as do conditions under which treatment-emergent side effects occur. c Not available, but incidence on placebo was reported to be greater than with the drug. d For depression + OCD + bulimia N 2444 ; , headache 21% + , dizziness 10% + , diarrhea 12% + , vomiting 3% + , asthenia 12% + , fever 2% + . e Studies or case reports in the literature. f Incidence was significantly lower in subsample of patients with depression other only.
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To faciltate interoperability, these new systems are required to be compatible with the ATN internet and upper layer protocol architectures. During the transition period it will be necessary to provide for the encoding of applications data into character based message formats for exchange over character oriented networks. 7.5.3 The transition to ATN is expected to start before the institutional issues involved in the administration of ATN have been fully agreed. The initial ATN routers should be procured by some States as COTS products, and upgraded as newer versions of the software are available to implement all of the necessary ATN options and parameters. Where ground-to-ground ATN is not available to support trials and demonstrations, such as the ATS Interfacility Data Communications AIDC ; application, the existing AFTN should be used to exchange message data units between those ATM systems. The transition from AFTN circuits to ATN internetworking will be on a circuit by circuit inter-domain connection basis, with time scales that can shall be quite independent of the other prior to any deployments of elements of the new CNS ATM systems. In order to migrate to ATN internetworking, there shall be clear identification of the domains to be interconnected. Furthermore, to establish a domain, States shall establish networks within their own domain to support inter-domain agreements. The determining factor will be the requirements placed on ATN communication services provided over the interconnected networks. Some AFTN circuits may be fully converted to ATN Internetworking well before any sole system use of ATN communication services occurs in air-ground systems of the new CNS ATM systems., Where other AFTN circuits may meet the service requirement by the continued use of AFTN well into the future, there will be a need to provide a gateway to communicate between the ATN and AFTN environments.
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Adeeko A, Forsyth DS, Casey V, Cooke GM, Barthelemy, J Cyr DG, Trasler JM, Robair B, and Hales BF 2003 ; Effects of in utero tributyltin chloride in the rat on pregnancy outcome. Toxicol Sci 74: 407 415. Bettin C, Oehlmann J, and Stroben E 1996 ; TBT-induced imposex in marine neo-gastropods is mediated by an increasing androgen level. Helgol Meeresunters 50: 299 317. Blair RM, Fang H, Branham WS, Hass BS, Dial SL, Moland CL, Tong, W Shi L, Perkins R, and Sheehan DM 2000 ; The estrogen receptor relative binding affin.
Fig 2 The effect of ketamine pretreatment on residual propofol concentration after incubation with rat hepatic microsomes. A ; Solid bar: microsomes from the control rats; hatched bar: microsomes from rats with ketamine pretreatment in absence and presence of orphenadrine. Each bar represents mean and SE. B ; Inhibition of PROD activity pmol resorufin min1 mg protein1 ; by orphenadrine in microsomes from ketamine-pretreated rats. Each point represents mean and SE. Data were analysed by ANOVA followed by Dunnett test to compare with the control group. There were three animals per group. * Value was significantly different from the control, P 0.05 and oseltamivir.
Through hard work and joint efforts by the whole nation, significant success has been achieved in the development of basic education, especially during the implementation of the Ninth Five-Year Plan 1996-2000 ; . By the end of 2000, the national average enrollment rate of primary school-age children had reached 99.1 per cent and the junior middle school gross enrollment rate had reached 88.6 per cent. The universalization of nine-year compulsory education.
Federdlon ci Clinical Research. Ballimore. MD. May 1992 ClIn Per 1992 354A ; . and at the 26th Annual Meeting ci the American Society ci Nephrology, Boon, MA, November 1993 J Soc Nephrol 1993A: 550 ; . 3Corr.ondence Tower. Universfly to Dr. P.S. Gasion, Division ci Nephrology, ci Alabama at BIrmlnam, Blrmlnam, 625 Tlnsley Harrison Al. 35294 and oxacillin.
CDD assigned to Giaconda, inter alia, all of its rights and title in and relating to the Invention; and Giaconda assumes the rights and obligations of CDD under the Deed of Assignment and Commercialisation that arise after the date of the Deed. The Deed of Assignment and Commercialisation was entered by CDD and the Inventors on 10 November 2003. This Deed assigned all rights and title in the Invention from the Inventors to CDD and Pharmatel. As a result of these Deeds: Pharmatel and Giaconda share equally the income and costs from the Invention; Pharmatel will be responsible for administering the patents relating to the Invention and any costs relating to the commercialisation of the Invention; Any dispute arising out of the commercialisation or maintenance of the Invention will be resolved by a decision of the majority of the Inventors. The term of the Assignment and Commercialisation Deed is 5 years after which time only the sharing of income and costs from the Invention will continue to apply.
Data were analyzed statistically by an analysis of variance followed by the Fisher's P least-significant difference multiple comparison test. The null hypothesis was rejected at the 0.05 level. ED50, EC50, and AD50 antagonist dose 50% ; values with 95% CLs were calculated by unweighted least-squares linear regression as described by Tallarida and Murray 1987 and oxaliplatin.
Since cGMP alters vasoconstrictor responses by diverse mechanisms, 30-32 such quantitative and qualitative differences may account for the differing effects of these cGMP-dependent vasodilators. Furthermore, we observed that ANP induced a transient, modest vasodilation of vessels preconstricted by elevated pressure Figure 3 ; . It possible that such high levels of ANP 10 , M ; may cause a transient increase in the rate of cGMP generation that exceeds steady-state levels. Divergent Effects of cGMP-Mediated Vasodilators on Receptor-Mediated Versus Depolarization-Induced Activation It has been reported that vascular smooth muscle responds differently to ANP and cGMP-mediated vasodilators when the preparations are precontracted with differing vasoconstrictors.3, 33, 34 ANP effectively relaxes the a-adrenergic- and angiotensin lI-stimulated aortic strips.24, 33-35 In contrast, contractions elicited by KCl-induced depolarization are refractory to ANP, 333 although submaximal KCIinduced contractions have been reported to be inhibited by ANP.34 35Furthermore, ANP has no effect on potassium-induced calcium influx, although it effectively inhibits norepinephrine-stimulated calcium influx.33 Similarly, nitroprusside inhibits norepinephrine-induced contractions, but not KCl-induced contractions of canine renal arterial strips.36 Taken together, these divergent effects of ANP and nitroprusside suggest that cGMP-mediated vasodilators selectively inhibit receptor-mediated vasoconstriction, whereas depolarization-induced vasoconstriction is refractory to cGMP.3 We have previously reported that ANP completely reverses norepinephrine-induced afferent arteriolar vasoconstriction.7 In the present study, we observed that nitroprusside and 8-bromo-cGMP also completely reverse norepinephrine-induced vasoconstriction of this vessel Figure 4 ; . In striking contrast, ANP, nitroprusside, and 8-bromo-cGMP were ineffective in inhibiting pressure-induced afferent arteriolar vasoconstriction. In concert with the formulation that pressure-induced vasoconstriction is mediated by depolarization-induced activation of voltagedependent calcium channels see below ; , our present findings are consistent with the premise that cGMPmediated vasodilators selectively inhibit receptormediated, but not depolarization-induced, vasoconstriction of the afferent arteriole. Mechanisms of Pressure-Induced Vasoconstriction The mechanisms mediating pressure-induced afferent arteriolar vasoconstriction have not been fully elucidated. Several lines of evidence indicate that pressure-induced vasoconstriction may be mediated by a pressure-induced membrane depolarization. Harder et a123 demonstrated that elevated pressure elicits membrane depolarization in isolated canine interlobular arteries and that verapamil prevented the pressureinduced vasoconstriction of this vessel. In the present.
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Mark et al, 32 it was shown that left ventricular outflow obstruction resulted in peripheral vasodilation. These data would suggest that left ventricular afferents mediated this response. Although we cannot definitively rule out a contribution of pulmonary afferents in this response33 on the basis of the two studies cited above, it does not appear that stimulation of pulmonary afferents is necessary for evoking reflex bradycardia during cardiac distension. Chemically sensitive afferents have been described throughout the left ventricular myocardium.1"34 PGs have been shown to stimulate these endings and to reverse the tachyphylaxis to bradykinin.35 Direct left circumflex coronary artery administration of PGI2 evokes a reflex bradycardia and sympathoinhibition by stimulation of left ventricular afferents.9'20 This reflex is mediated by an afferent vagal pathway, since application of lidocaine to the pericoronary nerves results in and oxandrolone.
C.L. Iwema and J.E. Schwob Department of Anatomy & Cell Biology, SUNY Health Science Center, Syracuse, NY 13210, USA.
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D Background and Aims Pistia stratiotes produces large amounts of calcium Ca ; oxalate crystals in specialized cells called crystal idioblasts. The potential involvement of Ca2 + channels in Ca oxalate crystal formation by crystal idioblasts was investigated. d Methods Anatomical, ultrastructural and physiological analyses were used on plants, fresh or xed tissues, or protoplasts. Ca2 + uptake by protoplasts was measured with 45Ca2 + , and the effect of Ca2 + channel blockers studied in intact plants. Labelled Ca2 + channel blockers and a channel protein antibody were used to determine if Ca2 + channels were associated with crystal idioblasts. d Key Results 45Ca2 + uptake was more than two orders of magnitude greater for crystal idioblast protoplasts than mesophyll protoplasts, and idioblast number increased when medium Ca was increased. Plants grown on media containing 150 mM of the Ca2 + channel blockers, isradipine, nifedipine or uspirilene, showed almost complete inhibition of crystal formation. When fresh tissue sections were treated with the uorescent dihydropyridine-type Ca2 + channel blocker, DM-Bodipy-DHP, crystal idioblasts were intensely labelled compared with surrounding mesophyll, and the label appeared to be associated with the plasma membrane and the endoplasmic reticulum, which is shown to be abundant in idioblasts. An antibody to a mammalian Ca2 + channel a1 subunit recognized a single band in a microsomal protein fraction but not soluble protein fraction on western blots, and it selectively and heavily labelled developing crystal idioblasts in tissue sections. d Conclusions The results demonstrate that Ca oxalate crystal idioblasts are enriched, relative to mesophyll cells, in dihydropyridine-type Ca2 + channels and that the activity of these channels is important to transport and 2004 Annals of Botany Company accumulation of Ca2 + required for crystal formation and oxaprozin.
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Description of Operation 1. Power-on sequence The CXA2025AS does not have an internal power-on sequence. Therefore, all IC operations are controlled by the set microcomputer I2C bus controller ; . 1 ; Power-on The IC is reset and the RGB outputs are all blanked. Hdrive starts to oscillate, but oscillation is at the maximum frequency 16kHz or more ; and is not synchronized to the input signal. Output of vertical signal VTIM starts, but Vdrive is DC output. Bus registers which are set by power-on reset are as follows. AGING1 0: All white output aging mode OFF AGING2 0: All black output aging mode OFF RON 0: Rch video blanking ON GON 0: Gch video blanking ON BON 0: Bch video blanking ON PICON 0: RGB all blanking ON VOFF 1: VDRIVE output stopped mode FHHI 1: H oscillator maximum frequency mode CD-MODE 0: Automatic selector mode of the countdown mode AKBOFF 0: AKB mode 2 ; Bus register data transfer The register setting sequence differs according to the set sequence. Register settings for the following sequence are shown as an example. Set sequence Power-on Degauss VDRIVE oscillation CXA2025AS register settings Reset status in 1 ; above. Reset status in 1 ; above. The CRT is degaussed in the completely darkened condition. The IC is set to the power-on initial settings. See the following page. ; A sawtooth wave is output to VDRIVE and the IC waits for the vertical deflection to stabilize. The HDRIVE oscillator frequency goes to the standard frequency around 15.734kHz ; . PICON is set to 1 and a reference pulse is output from Rout, Gout and Bout. Then, the IC waits for the cathode to warm up and the beam current to start flowing. Status register IKR is monitored. IKR 0: No cathode current IKR 1: Cathode current Note that the time until IKR returns to 1 differs according to the initial status of the cathode. RON, GON and BON are set to 1 and the video signal is output from Rout, Gout and Bout.
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From the Cleveland Clinic Florida, Ft Laudeniale, FL; Southwest Oncolngy Group Stahtical Center, Seattle; Fred Hutchinson Cancer Research Center, Seattle, WA; St Jude Children's Research Hospital, ho Memphis, nV; O i State University Health Center, Columbus, OH; Louisiana State University Medical Center, New Orleans, LA; Wichita C o m Clinical OncologyProgram, Wichita, KS; University of California at Davis, Sacramento, CA; Allegheny Health, Education and Research, Pittsburgh, PA; and Johns Hophim University, Baltimore, MD. Submitted August 15, 1995; accepted May 31, 19%. Supported in part by the following Public Health Service Cooperative Agreement grants awarded by the National Cancer Institute, Department of Health and Human Services: CA38926, CA32102, CA04920, CA58658, CA04919, CA35431, CA58861, CA16385, CA46441, CA35117, CA12213, CA46136, CA58882, CA35128, CA3.5192, CA42028, CA13612, CA35261, CA27057, CA42777, CA32734, CA46282, CA58686, CA58416, CA35119, CA35090, CA20319, CA37981, CA351 78, CA12644, CA45807, CA35261, CA28862, CA35262, CA461 13, CA22433, CA58415, CA351 76, CA35281, CA45560, and CA52654. Address reprint requests to Southwest Oncology Group SWOG8600 ; . Operations Office, 14980 Omicron Dr, San Antonio, 7X 78245-3217. Zie publication cosis of this article were defrayed in part by page charge payment. This article must therefore be hereby marked "advertisement" i accordance with 18 U.S.C. section 1734 solely to n indicate this fact. 0 1996 by The American Society of Hematology. 0006-497I 96 8808-0032.00 0.
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FIG. 4. A schematic diagram showing potential endocannabinoid signaling in blastocyst activation and implantation. Anandamide and 2-AG are the major endocannabinoids that interact with G protein-coupled cannabinoid receptors, CB1 and CB2. Regulated levels of endocannabinoids in the receptive uterus and CB1 in activated blastocysts at the time of implantation are beneficial for implantation, whereas higher levels are detrimental to this process. Because COX-2 is expressed in the uterus at the site of implantation and because anandamide and 2-AG can serve as substrates for either COX-2 or FAAH, the proposal suggests that uterine endocannabinoids are tightly regulated by the coordinated activity of FAAH and COX-2 in the uterus during early pregnancy. Evidence suggests that regulated uterine levels of endocannabinoids and blastocyst CB1 play a physiological role in synchronizing blastocyst competency with uterine receptivity for implantation. Tr, Trophectoderm; ANA, anandamide; CB1, brain-type cannabinoid receptor; FAAH, fatty acid amide hydrolase; IS, implantation site; Inter-IS, interimplantation site.
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The new trends in landscaping are now creating a new, more natural look for waterfront properties. Recognizing that traditional development patterns have negatively affected the health of our waterbodies, landscape professionals are maintaining shoreline vegetation on newly developed lots. They are also adding a new mix of vegetation and color to old lots to create a fresh look. In both cases, these new trends help to protect water quality, provide wildlife a refuge and ultimately maintain or increase property values. The main body of this manual is intended to help you look at your waterfront property in a new way. line vegetation and displaced wildlife, as in this First we will explain what a vegetated buffer is lakefront property. and how it benefits the health of your water body. Source: BRPC archive, 2002. Next we will examine a few examples of buffers at work. Then we will offer creative new ways to help you landscape your shoreline without losing your yard, your view or your access to the water. An extensive list of native plants, which also includes the conditions soil type, sunlight level, etc. ; under which each plant will grow the best, is included in Appendix B.
Inhalers pressurized with chlorofluorocarbons.4 Regulatory guidelines from the US Food and Drug Administration demand consistent dose delivery.1 Multiple studies5 have reviewed drug delivery with these DPIs, which appear to be comparable to metered-dose inhalers. However, these studies are almost always tested under clinical research conditions and not field conditions. Although the manufacturer recommends storage and transport conditions that would obviate the decrease in powder delivery seen in the present studies, the clinical observation that patients returned capsules that appear similar to those heated in the laboratory again demonstrates that actual clinical conditions and use may differ from clinical trial conditions and use. Arizona during the summer is a particularly harsh climate with temperatures that are often as high as 115F 46C ; . Temperatures in parked cars have been reported as high as 70C 158F ; . At this temperature, we observed powder clumping and capsule distortion with formoterol.6 Not surprisingly, we found similar high temperatures in mailboxes. This may be a peculiarity of environments with extremes in temperature such as Arizona. In support of this concept, we could find no reports in PubMed using a search strategy of DPI and temperature that reported a decrease in drug delivery with extremes in.
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E878.3 Surgical operation with formation of external stoma Colostomy Cystostomy Duodenostomy Gastrostomy Ureterostomy E878.4 Other restorative surgery E878.5 Amputation of limb s ; E878.6 Removal of other organ partial ; total ; E878.8 Other specified surgical operations and procedures E878.9 Unspecified surgical operations and procedures E879 Other procedures, without mention of misadventure at the time of procedure, as the cause of abnormal reaction of patient, or of later complication E879.0 Cardiac catheterization E879.1 Kidney dialysis E879.2 Radiological procedure and radiotherapy Excludes: radio-opaque dyes for diagnostic x-ray procedures E947.8 ; E879.3 Shock therapy Electroshock therapy Insulin-shock therapy E879.4 Aspiration of fluid Lumbar puncture Thoracentesis E879.5 Insertion of gastric or duodenal sound E879.6 Urinary catheterization E879.7 Blood sampling E879.8 Other specified procedures Blood transfusion E879.9 Unspecified procedure ACCIDENTAL FALLS E880-E888 ; Excludes: falls in or from ; : burning building E890.8, E891.8 ; into fire E890.0-E899 ; into water with submersion or drowning ; E910.0-E910.9.
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Do not use orphenadrine if: you are allergic to any ingredient in orphenadrine you have glaucoma, stomach or intestinal blockage, stomach ulcers, a blockage of your bladder, certain problems with your esophagus eg, decreased esophageal motility or severe irritation caused by reflux ; , enlargement of the prostate, ulcerative colitis, or myasthenia gravis contact your doctor or health care provider right away if any of these apply to you.
Chlorzoxazone cyclobenzaprine hcl methocarbamol orphenadrine citrate SKELAXIN * Inj. Drugs for Arthritis EUFLEXXA [INJ] Non-Steroidal AntiInflammatory Agents CELEBREX [ST] diclofenac sodium etodolac ibuprofen indomethacin meloxicam nabumetone naproxen Salicylates & Related Drugs choline mag trisalicylate diflunisal salsalate NUTRITION & BLOOD MODIFIERS Antiplatelet Drugs cilostazol dipyridamole PLAVIX Blood Detoxicants lactulose RENAGEL Oral Anticoagulants warfarin Therapeutic Vitamins & Minerals folic acid PHOSLO OBSTETRICAL & GYNECOLOGICAL MEDICATIONS Androgen Drugs TESTIM Contraceptives NOTE: Coverage based on benefit design. apri aranelle aviane camila cesia cryselle enpresse errin jolivette junel, fe kariva kelnor leena lessina levora low-ogestrel lutera microgestin, fe mononessa continued.
Several climate reconstructions have been made for the last millenium, with different and sometimes controversial results which mainly put forward the natural variability of global temperature GT ; Esper, 2002, Broecker, 2001 ; . These reconstructions, that extend the instrumental record backwards in time, are based on several proxy records of climate change contained mainly in naturally occurring archives, such as tree-rings, ice core layers, boreholes and glacial advances. Also, these GT reconstructions employ different statistical techniques in the calibration and backward extrapolations of climate records. This diversity of information and methods, in which climate reconstructions are recently based, produce a wealth of alternatives, but also possible significant differences in the results. It therefore appears necessary to test another alternatives for climate reconstruction which could give us more elements to evaluate and compare all of them, and this is what this contribution intends to do by presenting a new GT record for the last millenium recently reconstructed SnchezSesma, 2003a ; . This presentation will mainly consider the basis and results of a novel contribution to global climate reconstructions. This new reconstruction employs a single proxy record and a simple calibration procedure which emphasize low frequency climate oscillations. A comparison with previous GT reconstructions obtained by Mann et al, 1999 ; , Esper et al, 2002, and Mann et al, 2003 ; has been made Snchez-Sesma, 2003b ; . This comparison has detected coincidences and also differences between the GT reconstructed records, confirming the occurrences of two well documented climate extremes known as the Medieval Warm Period MWP ; and the Little Ice Age LIA ; , and giving elements to asses the natural contribution to the recent global warming. REFERENCES. Broecker, WS, 2001, Was the medieval Warm Period Global?, Science, 292, 1497. Esper J, Cook ER, and Schweingruber FH, 2002, Low-Frequency Signals in Long Tree-Ring Chronologies for Reconstructing Past Temperature Variability, Science, 295 5563 ; , 2250-2253. Mann ME, Bradley RS, and Hughes MK, 1999. Northern Hemisphere Temperatures During the Past millenium: Inferences, Uncertainties, and Limitations. Geophysical Research Letters, 26, 759. Mann ME, Rutherford S, Bradley RS, and Hughes MK, and Keimig FT. 2003. OptimalSurface Temperature Reconstructions Using Terrestrial Borehole Data. Journal Geophysial Research Atmospheres 108, D7. Snchez-Sesma J. 2003a. Analysis and Synthesis of Climate Change, PhD Thesis, UNAM, Mexico. Snchez-Sesma J. 2003b. Global Temperature GT ; Reconstructed Records for the Last Millenium: A Review. Submitted to the NASA-CRCES-IPRC Workshop on Decadal Climate Variability to be held in Kona Hawaii, February, 2004.
38. Tofovic SP, Salah EM, Dubey RK, Melhem MF, and Jackson EK. Estradiol metabolites attenuate renal and cardiovascular injury induced by chronic nitric oxide synthase inhibition. J Cardiovasc Pharmacol 46: 2535, 2005. Tomiyoshi Y, Sakemi T, Aoki S, and Miyazono M. Different effects of castration and estrogen administration on glomerular injury in spontaneously hyperglycemic Otsuka Long-Evans Tokushima fatty OLETF ; rats. Nephron 92: 860 867, Wells CC, Riazi S, Mankhey RW, Bhatti F, Ecelbrager C, and Maric C. Diabetic nephropathy is associated with decreased circulating estradiol levels and imbalance in the expression of renal estrogen receptors. Gender Med 2: 227237, 2005. Zaoui P, Cantin JF, Alimardani-Bessette M, Monier F, Halimi S, Morel F, and Cordonnier D. Role of metalloproteases and inhibitors in the occurrence and progression of diabetic renal lesions. Diabetes Metab 26 Suppl 4: 2529, 2000.
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